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-Hypertension [1].*Correspondence: j.chen@vumc.nl ^ Deceased 1 Diabetes Center/Department of腾龙公司【微kokang111】
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发布于:2021-5-21 11:15:55  访问:10 次 回复: 篇
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Hypertension [1].*Correspondence: j.chen@vumc.nl ^ Deceased 1 Diabetes Center/Department of
However, underlying mechanisms?The Author(s) 2017. This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and MedChemExpress 848141-11-7 reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/ publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.Chen et al. Cardiovasc Diabetol (2017) 16:Page 2 offor this effect remain to be elucidated. Small scaled studies suggested enhanced endothelial function and increased perfusion by GLP-1 [9, 10]. Moreover, shift towards augmented glucose metabolism has favorable effects on cardiac energetics [11]. Both phenomena have important prognostic relevance [12, 13]. At present, effects of GLP-1 receptor CS-1695 web agonists (RA) on myocardial perfusion and energetics in T2DM patients with PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/11097623 LV CS-1761 systolic dysfunction are unclear. In view of the diabetes pandemic and the associated high risk of HF, bloodglucose-lowering agents that can be prescribed safely are of great importance. Therefore, the aim of this study was to examine effects of GLP-1RA on cardiac function, myocardial perfusion, and energetics in T2DM patients with LV systolic dysfunction compared to insulin glargine.breakfast and study medication, prior to randomization, and patients underwent follow-up measurements after 26 weeks of treatment.OutcomesThe primary outcome in the current study was effects on PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/19815599 LVEF after 26-week treatment of exenatide versus insulin glargine in T2DM patients with LV systolic dysfunction. Secondary outcomes were differences in myocardial perfusion and energetics in T2DM patients with LV systolic dysfunction versus healthy BMI-matched healthy controls, and effects on myocardial perfusion and energetics after 26-week treatment of exenatide versus insulin glargine in T2DM patients with LV systolic dysfunction.CMRMethodsParticipantsT2DM patients with LV dysfunction, LV ejection fraction (LVEF) < 50 [as documented in the medical records, measured using echocardiogram, radionuclide angiogram or cardiovascular magnetic resonance imaging (CMR)], above 18 years, body mass index (BMI) of 25?0 kg m-2, hemoglobin A1C (HbA1c) of 6.5?0.0 (48?6 mmol mol-1), were randomized, at an allocation ratio of 1:1, open-label, to exenatide or insulin glargine on top of ongoing use of oral glucose-lowering agents (metformin or metformin and sulfonylurea) after a run-in period of 10 weeks. Exclusion criteria were renal or liver impairment, malignancy, cardiovascular events <3 months, insulin, thiazo.Hypertension [1].*Correspondence: j.chen@vumc.nl ^ Deceased 1 Diabetes Center/Department of Internal Medicine, VU University Medical Center, de Boelelaan 1117, 1081 HV Amsterdam, The Netherlands Full list of author information is available at the end of the articleThe optimal bloodglucose-lowering therapy in T2DM patients and HF is still under debate. Multiple agents [2?] have been linked to cardiovascular events. Other agents [5, 6] have been associated with a lower risk of cardiovascular events. Preliminary studies have shown recovery of left ventricular (LV) function during glucagon-like peptide-1 (GLP-1) administration in HF patients irrespective of the diabetic status [7, 8].
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